POTS names what your heart does when you stand. Our team looks for why it happens. Start with the racing heart itself, then the conditions it gets mistaken for, the symptoms that come with it, what helps and what recovery looks like.
Your heart races when you stand because your brain is losing blood flow, a measurable regulation failure, not anxiety. Here's what's actually happening.
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What causes the high heart rate in POTS? The heart speeds up to rescue blood flow to your brain. Why it happens, what beta blockers reveal and what to measure.
POTS isn't one thing. Understanding the three subtypes, neuropathic, hyperadrenergic, and hypovolemic, is the first step toward treatment that works.
Hyperadrenergic POTS (hyperPOTS) is POTS with a norepinephrine surge on standing. See the symptoms, how it's tested, what beta blockers miss and what to check.
Orthostatic intolerance means symptoms when you stand that ease when you lie down. Symptoms, causes, and the types: orthostatic hypotension, POTS and OCHOS.
If your blood pressure looks normal and your heart rate doesn't quite qualify, you've been told nothing is wrong. Here's what the standard test misses.
The autonomic nervous system isn't an on/off switch. Learn why 'sympathetic dominance' is misleading and how specific pathways drive POTS symptoms.
POTS or anxiety? How to tell which comes first, why POTS is so often misdiagnosed as anxiety, what testing shows, and a case where the body led.
FND and POTS can show up together. What FND is, how it differs from POTS, why standing can make FND symptoms worse, and what a tilt test can show.
Why so many POTS cases get dismissed, why clean labs don't mean you're fine, and how a brain-first approach finds what standard workups miss.
A POTS diagnosis can be a relief, but for many the treatments still don't work. Here's why, and what understanding your specific mechanism changes.
Small fiber neuropathy shows up in a quarter to half of POTS patients tested. How SFN drives dysautonomia, why a skin biopsy can miss it, and what improves it.
How MCAS and dysautonomia connect: what histamine does to blood flow and heart rate in POTS, how to tell the 2 apart, and what helps when both flare.
Can B6 toxicity cause POTS? Learn about vitamin B6 and autonomic nerves, how to start moving again with POTS, and hypoplastic vertebral arteries.
Does deconditioning cause POTS, or POTS cause deconditioning? What bed rest does to the heart, POTS vs being out of shape, and what exercise can and can't do.
Blood pooling is blood collecting in your legs, feet or hands when you're upright. What it looks like, when it's normal, and what causes it in POTS.
For most people with POTS, low blood volume is not the core problem. Here is why salt and water hit a ceiling, and what fluid dumping really means.
Why POTS and dysautonomia cause nausea, bloating, and gastroparesis. The gut isn't broken; the brain's control of it is. The autonomic-GI connection.
What causes brain fog, why it can get worse when you stand or drive, and how to find which link in your brain's supply chain is weak.
Ear fullness, popping, or a heartbeat in your ear with POTS? It often traces to how blood drains from your head, not just your eustachian tubes.
Feeling detached from reality with POTS or dysautonomia? Derealization is often a blood-flow and vestibular signal, not just anxiety. What it means.
Why exercise feels so hard with POTS: the symptoms, what research shows about the heart and fitness, what our team finds in the brain, and what helps.
How much salt and water actually helps POTS, where the 2 to 3 liter advice stops working, and the signs your real problem is blood distribution.
POTS medications manage the compensation. Neurological rehab targets the regulation failure. Understand where each approach works and where it falls short.
Do SSRIs help POTS? What the one randomized SSRI trial found, why SNRIs and ADHD drugs can raise standing heart rate, and how to talk with your prescriber.
IV saline helps ME/CFS and dysautonomia patients feel better temporarily. But if the problem is blood flow distribution, saline is a patch, not a fix.
Exercise advice fails POTS patients because it ignores orthostatic blood flow. Deconditioning isn't the cause. Fix cerebral perfusion first.
Why do so many dysautonomia treatments fail? Both conventional and functional medicine miss the one core problem: the brain is the common thread.
Vagus nerve stimulation for POTS misses the point. The vagus is mostly sensory, paired antagonism is a myth, and the real driver of dysautonomia differs.
POTS and dysautonomia treatment should recalibrate your nervous system, not just manage symptoms. Targeting the lynchpin unlocks recovery.
POTS is a brain regulation problem, not a heart rate problem. Mechanism-based neurological evaluation identifies what's actually driving symptoms.
Peripheral nerve stimulation lets dysautonomia patients train the brain without crashing. How PNS restores proprioception and cerebral blood flow.
Sleep is actively restorative. Learn why sleep is the engine of neuroplasticity and recovery in dysautonomia and POTS.
What intensive POTS treatment involves: cerebral blood flow testing, vestibular rehab, and neurological rehabilitation. What to expect in 4 to 7 days.
Can POTS be cured? Many recover significantly or fully. 'Cure' means recalibrating a nervous system that lost its precision, and research shows it can.
How to know if your POTS is actually improving using cerebral blood flow data, not just symptoms. A case study shows why BP meds may worsen perfusion.
A 27-year-old with brain fog and POTS after a concussion recovered cerebral blood flow and resolved heart rate instability in two weeks.
A patient with two years of POTS recovered in one month through targeted neural rehab: cerebral blood flow testing, tilt progression, and retinal slip.
No study we found counts how many people with POTS stay in a wheelchair. See one 8-year case: what our team found, what we did and what changed.
Case study: A college-aged EDS and POTS patient went from missing school and extreme fatigue to getting back to life in under a month.
POTS case study: A 21-year-old athlete went from housebound to coaching baseball and taking classes again. Neuropathy findings and CO2-guided rehab.
A 21-year-old with POTS and gastroparesis improved digestion, heart rate, and sensation through neural rehab, without any GI-specific treatment.
A 15-year-old diagnosed with POTS wasn't actually a POTS case. See the real diagnosis, brain-based treatment, and full recovery including return to school.
For how our team evaluates and treats POTS, see the POTS page. Other forms of dysautonomia are on the dysautonomia page.
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