Educational articles about neurological conditions, diagnostic approaches, and what actually works for complex cases.
Your POTS diagnosis told you what's happening. It didn't tell you why. Understanding the difference is the key to actually getting better.
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Heart rate is only part of the story. Measuring actual blood flow to the brain reveals what's really happening when you stand up.
Eye tracking isn't just for research labs. It's one of the most powerful tools we have for assessing brain function.
The old advice was to rest until symptoms resolve. For many patients, that never happens. Here's what actually works.
Not all dizziness is the same. Identifying which component of your vestibular system is affected determines what treatment will work.
A positive tilt table test confirms POTS. But it doesn't tell you why. Here's what we test to find the actual mechanism.
Medication manages the pain. But migraine is more than pain — it's a neurological event. Treating it that way opens new options.
Your neck does far more than hold your head up. When cervical joints aren't working properly, the consequences reach your brain.
Neuropathy doesn't mean your nerves are dead. Understanding the type you have changes everything about treatment and prognosis.
Brain fog is a symptom, not a condition. Treating it directly without understanding the mechanism is why so many people stay stuck.
When "just exercise more" makes everything worse, the problem isn't effort. It's what the brain is doing with blood flow during exertion.
Structural issues CAN change cerebral perfusion — but only if that's actually the impediment to blood flow in the first place.
IV fluids make you feel better for a few hours. But if the problem isn't total volume, why would adding more volume be the answer?
ME/CFS isn't just being tired. It's a real, measurable neurological condition — and diagnosing it properly starts with finding the mechanism.
The crash has a mechanism. Understanding that mechanism is how you stop post-exertional malaise from running your life.
FND is one of the most misunderstood diagnoses in neurology. Here's what it actually means and why it's not "all in your head."
Normal blood pressure doesn't mean normal brain blood flow. Here's why that gap exists and what it means for your symptoms.
You've tried the medications, the salt, the compression stockings. If they're not working, the problem might not be the treatment — it might be the diagnosis.
Everyone talks about deficiency. Almost nobody talks about toxicity. But too much of certain supplements can make your autonomic symptoms worse.
POTS isn't just a heart rate problem. It's what happens when your brain can't properly coordinate the systems that keep you upright.
The "floppy veins" explanation is everywhere. But the actual mechanism is more interesting — and more treatable — than that.
Your heart rate spikes when you stand up. Everyone focuses on slowing it down. But the tachycardia isn't the problem — it's a symptom.
"Just exercise more" is the most common advice. But is deconditioning really the cause — or a consequence of something else going wrong?
Sleep isn't rest — it's a highly regulated, actively restorative process. If your brain is already struggling, it changes everything about how you recover.
When we stimulate the brain in the right way, we can watch the autonomic outputs change — in real time, on every measure we track.
A 21-year-old with dysautonomia and gastroparesis improved digestion, heart rate, and sensation through neural rehabilitation — without any GI-specific treatment.
How we used cerebral blood flow testing, tilt angle progression, and retinal slip exercises to rebuild orthostatic tolerance from the ground up.
A college-aged patient with Ehlers-Danlos Syndrome and POTS went from missing school and extreme fatigue to getting back on track.
A 21-year-old college athlete couldn't drive, shop for groceries, or attend school. Here's how we approached his case — and what happened.
A real case study walking through the tilt test data — what the blood flow numbers actually show and why one side dropped and the other didn't.
Autonomic hyperactivity isn't just "too much adrenaline." It's what happens when the brain stops providing the inhibition that keeps the system in check.
A 15-year-old with severe migraines, dizziness, and air hunger had been diagnosed and treated for POTS. The real issue was in the brain.
Dizziness, heart rate spikes to 170, sweaty hands, nausea, and broken sleep. Here's how we found the problem in the eyes and rebuilt it step by step.
After 8 years of POTS and daily wheelchair use, a patient left the chair behind in just a few weeks through targeted brain rehabilitation.
Dr. Keiser explains how stimulant medications affect POTS symptoms, why your body compensates with energy mobilization, and how sunlight plays a role.
Loads of people with POTS are diagnosed with anxiety as a brush-off. Here's how we figure out which is actually driving the symptoms.
Why standard workups miss the real problem in POTS, and why clean MRIs and normal blood work don't mean nothing is wrong.
Heart rate isn't the only measure of progress. Dr. Keiser explains what real POTS improvement looks like beyond the numbers.
A complete breakdown of orthostatic intolerance — what causes it, how it differs from POTS, and why blood flow to the brain matters more than heart rate.
Case study showing measurable POTS improvement after just two weeks of targeted neurological rehabilitation.
Dr. Keiser explains what functional neurology actually is, how it differs from traditional neurology, and what kind of patients benefit most.
Your brain controls its own blood supply separately from the rest of your body. When that system fails, standard treatments can't reach the problem.
Getting a tilt table test? Dr. Keiser clears up the biggest misconceptions and explains what a good tilt test should actually measure.
Mast cell activation and POTS are connected through specific sympathetic pathways. How histamine drives blood pooling and autonomic compensation.
POTS treatment should recalibrate your nervous system, not just manage symptoms. Why targeting the lynchpin unlocks recovery.
CFS, POTS, and ME/CFS syndrome labels describe symptoms, not causes. Moving from syndrome to mechanism is the key to effective treatment.
Why "sympathetic dominance" is misleading and how specific autonomic pathways — not an on/off switch — drive POTS symptoms.
The cerebellum regulates autonomic reflexes. When it's not calibrating properly, POTS and dysautonomia symptoms follow.
Vagus nerve stimulation for POTS misses the point. The vagus nerve is mostly sensory, and "paired antagonism" is a myth.
Exercise advice fails POTS patients because it ignores orthostatic blood flow. Fix cerebral perfusion first, then build capacity.
Mal de Debarquement Syndrome explains why passive motion feels better. The sensory mismatch driving your symptoms.
POTS heart rate spikes are compensatory, not the root problem. Cerebral blood flow, not tachycardia, is the real issue.
Neck stiffness with dysautonomia isn't random. The neurological connection between your cervical spine, vestibular system, and autonomic symptoms.
POTS fatigue and brain fog aren't laziness. Reduced cerebral blood flow starves your brain of fuel. Here's what testing reveals.
Post-concussion symptoms that persist involve disrupted cerebral blood flow and cerebellar dysfunction. The brain mechanisms behind lingering recovery.
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